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Renin Angiotensin Mechanism.

Biotransformation of Drugs: Phase I Reactions.

 

  • These reactions generally precede phase II reactions and include oxidative, reductive and hydrolytic reactions. 

  • Generally the polar groups are added to the xenobiotics.

  • Called “Non Synthetic Reactions” as the product is not totally altered.

  • The primary objectives phase I reactions are,

    • 1. Increase in hydrophilicity

    • 2. Reduction in stability

    • 3. Facilitation of conjugation (Phase II).

  • Outcome of the Phase I reactions may be active, more active or inactive metabolites.

  • These reactions are termed non-synthetic reactions, and include oxidation, reduction, hydrolysis, cyclization and decyclization reactions. 

  • These reactions are carried out mostly by mixed function oxidases, usually involving CYP450 and occur in the liver. 

  • In these reactions, a polar group is either introduced or unmasked if

  • already present. 

  • These reactions are succeeded by Phase II reactions. 

  • Most of the Phase I products are not eliminated directly; instead they undergo Phase II reactions. 

  • Various Phase I reactions are as follows, with several examples:

  1. Oxidation:

  1. Oxidation of aromatic moieties.

  2. Oxidation of olefins.

  3. Oxidation at benzylic, allylic carbon, carbon atoms α to carbonyl and imines.

  4. Oxidation at aliphatic and alicyclic carbon.

  5. Oxidation involving carbon-heteroatom systems: 

    • Carbon-nitrogen systems:

      • aliphatic and aromatic amines; 

      • N-dealkylation, 

      • oxidative deamination, 

      • N-oxide formation, 

      • N-hydroxylation.

    • Carbon-oxygen systems:

      • O-dealkylation

    • Carbon-sulfur systems:

      • S-dealkylation, 

      • S-oxidation, and 

      • Desulfurization

  6. Oxidation of alcohols and aldehydes • 

  7. Other miscellaneous oxidative reactions 

B) Reductive Reactions:

  1. Reduction of aldehydes and ketones

  2. Reduction of nitro and azo compounds

  3. Miscellaneous reductive reactions

C) Hydrolytic Reactions:

  1. Hydrolysis of esters and amides

  2. Hydration of epoxides and arene oxides by epoxide hydrolase.

Oxidation Reactions.

1) Oxidation of aromatic moieties:

  • Monosubstituted benzene derivatives can be hydroxylated at ortho-, meta- or para-positions but para-hydroxylated product is most common, 

  • e.g. conversion of acetanilide to paracetamol, and phenylbutazone to oxyphenbutazone.

2) Oxidation of Olefins:

  • A better known example of olefinic oxidation is conversion of carbamazepine to carbamazepine-10,11-epoxide; the latter is converted to corresponding trans-10,11-dihydrodiol.

3) Oxidation of Benzylic Carbon Atoms:

  • Carbon atoms attached directly to the aromatic rings (benzylic carbon atoms) are hydroxylated to corresponding carbinols. 

  • If the product is a primary carbinol, it is further oxidized to aldehydes and then to carboxylic acids, e.g. tolbutamide. 

  • A secondary carbinol is converted to ketone.

4) Oxidation of Allylic Carbon Atoms:

  • Carbon atoms adjacent to olefinic double bonds (are allylic carbon atoms) also undergo hydroxylation in a manner similar to benzylic carbons, e.g. hydroxylation of hexobarbital to 3'-hydroxy hexobarbital.

5) Oxidation of Carbon Atoms Alpha to Carbonyls and Imines:

  • Several benzodiazepines contain a carbon atom (C-3) alpha to both carbonyl (C=O) and imino (C=N) functions which readily undergo hydroxylation, e.g. diazepam.

6) Oxidation of Aliphatic Carbon Atoms (Aliphatic Hydroxylation):

  • Alkyl or aliphatic carbon atoms can be hydroxylated at two positions - at the terminal methyl group (called as -oxidation) and the penultimate carbon atom (called as -1 oxidation) of which the latter accounts for the major product, e.g. valproic acid. 

7) Oxidation of Alicyclic Carbon Atoms (Alicyclic Hydroxylation):

  • Cyclohexane (alicyclic) and piperidine (non aromatic heterocycle) rings are commonly found in a number of molecules, e.g. acetohexamide and minoxidil respectively. 

  • Such rings are generally hydroxylated at C-3 or C-4 positions.

8) Oxidation of Carbon-Nitrogen Systems:

  • A) N-Dealkylation:

    • Alkyl groups attached directly to nitrogen atom in nitrogen bearing compounds are capable of undergoing N-dealkylation reactions, e.g. secondary and tertiary aliphatic and aromatic amines, tertiary alicyclic amines and N-substituted amides and hydrazines.

    • Tertiary nitrogen is more rapidly dealkylated in comparison to secondary nitrogen because of its higher lipid solubility. Thus, one alkyl from a tertiary nitrogen compound is removed rapidly and the second one slowly.

  • B) Oxidative Deamination: 

    • Like N-dealkylation, this reaction also proceeds via the carbinolamine pathway but here the C-N bond cleavage occurs at the bond that links the amino group to the larger portion of the drug molecule.

  • C) N-Oxide Formation:

    • N-oxides are formed only by the nitrogen atoms having basic properties. 

    • Thus, amines can form N-oxides but amides cannot. 

    • Generally, the tertiary amines yield N-oxides.

  • N-Hydroxylation:

    • Converse to basic compounds that form N-oxides, N-hydroxy formation is usually displayed by non-basic nitrogen atoms such as amide nitrogen, e.g. lidocaine.

    • N-hydroxylation of amides often leads to generation of chemically reactive intermediates capable of binding covalently with macromolecules, e.g. paracetamol. 

    • Paracetamol is safe in therapeutic doses since its reactive metabolite iminoquinone is neutralized by glutathione

    • However, in high doses, the glutathione level becomes insufficient and significant covalent tissue binding thus occurs, resulting in hepatotoxicity.

9) Oxidation of Carbon-Sulfur Systems:

  • A)  S-Dealkylation:

    • The mechanism of S-dealkylation of thioethers (RSR’) is similar to N-dealkylation i.e. it proceeds via -carbon hydroxylation. 

    • The C-S bond cleavage results in the formation of a thiol (RSH) and a carbonyl product, e.g. 6-methyl mercaptopurine.

  • B) Desulphurization:

    • This reaction also involves cleavage of the carbon-sulfur bond (C=S or thiono). 

    • The product is the one with a C=O bond. 

    • Such a desulphurization reaction is commonly observed in thioamides (RCSNHR’) such as thiopentone.

  • C) S-Oxidation:

    • Apart from S-dealkylation, thioethers can also undergo S-oxidation reactions to yield sulfoxides which may be further oxidized to sulfones (RSO2R). 

    • Several phenothiazines, e.g. chlorpromazine, undergo S-oxidation.

10) Oxidation of Carbon-Oxygen Systems:

  • O-Dealkylation: This reaction is also similar to N-dealkylation and proceeds by α-carbon hydroxylation to form an unstable hemiacetal intermediate which spontaneously undergoes C-O bond cleavage to form alcohol  and a carbonyl moiety.

11) Oxidation of Alcohol, Carbonyl and Carboxylic Acid:

  • These reactions are mainly catalyzed by non-microsomal enzymes, dehydrogenases. 

  • Primary and secondary alcohols and aldehydes undergo oxidation relatively easily but tertiary alcohols, ketones and carboxylic acids are resistant since such a reaction involves cleavage of C-C bonds.

  • Primary alcohols are rapidly metabolized to aldehydes (and further to carboxylic acids) but oxidation of secondary alcohols to ketones proceeds slowly.

12) Miscellaneous Oxidative Reactions:

  • A) Oxidative Aromatisation/Dehydrogenation: 

  • B) Oxidative Dehalogenation:

Reduction Reactions.

  • Bio-Reductions are also capable of generating polar functional groups such as hydroxy and amino which can undergo further biotransformation or conjugation. 

  • A number of reductive reactions are exact opposite of oxidation. For example:

  • Alcohol dehydrogenation ↔ Carbonyl reduction

  • N-Oxidation ↔ Amine oxide reduction.

  1. Reduction of Carbonyls (Aldehydes and Ketones):

  • Few aldehydes undergo reduction because such a reaction is usually reversible, and secondly, they are susceptible towards oxidation which yields more polar products. 

  • Several ketones undergo reduction as it results in more polar metabolites. 

  • Reduction of aldehydes and ketones yields primary and secondary alcohols respectively. 

  • The reaction is catalyzed by non-microsomal enzymes called aldo-keto-reductases.

  1. Reduction of Alcohols and Carbon-Carbon Double Bonds:

  • Reduction of norethindrone, an , -unsaturated carbonyl compound, results in both reduction of C=C double bond and formation of alcohol.

  1. Reduction of N-compounds (Nitro, Azo and N-Oxide):

  • The N-containing functional groups that commonly undergo bioreduction are nitro, azo and N-oxide. 

  • It is the reverse of oxidation.

  1. Miscellaneous Reductive Reactions:

  • A) Reductive Dehalogenation:

    • This reaction involves replacement of halogen attached to the carbon with the H-atom, e.g. halothane.

  • B) Reduction of Sulfur Containing Functional Groups: 

    • An example of S-S reductive cleavage is disulfiram.

HYDROLYTIC REACTIONS.

  • The hydrolytic enzymes that metabolize xenobiotics are the ones that also act on endogenous substrates. 

  • Moreover, their activity is not confined to the liver as they are found in many other organs like kidney, intestine, etc.

  • A number of functional groups are hydrolysed viz. esters, ethers, amides, hydrazides, etc.

  1. Hydrolysis of Esters and Ethers:

  • Esters on hydrolysis yield alcohol and carboxylic acid. 

  • The reaction is catalyzed by esterases.

  • Aromatic esters are hydrolysed by arylesterases and aliphatic esters by carboxylesterases.

  1. Hydrolysis of Amides (C-N bond cleavage):

  • Amides are hydrolysed slowly in comparison to esters. 

  • The reaction, catalyzed by amidases, involves C-N cleavage to yield carboxylic acid and amine.

  1. Hydrolytic Cleavage of Non-aromatic Heterocycles:

  • Non Aromatic heterocycles also contain amide functions, e.g. lactams (cyclic amides), e.g. Penicillins.

  1. Hydrolytic Dehalogenation:

  • Chlorine atoms attached to aliphatic carbons are de-halogenated easily, e.g. DDT.

Commonly Asked Questions.

  1. Define Biotransformation of drugs. Write a short note on “Phase I Reactions”.

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